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Pathogenesis of Neuropathic Pain = Diagnosis and Treatment /
Record Type:
Language materials, printed : Monograph/item
Title/Author:
Pathogenesis of Neuropathic Pain/ edited by Daryl I. Smith, Hai Tran.
Reminder of title:
Diagnosis and Treatment /
other author:
Smith, Daryl I.
Description:
XVII, 289 p. 42 illus. in color.online resource. :
Contained By:
Springer Nature eBook
Subject:
Anesthesiology. -
Online resource:
https://doi.org/10.1007/978-3-030-91455-4
ISBN:
9783030914554
Pathogenesis of Neuropathic Pain = Diagnosis and Treatment /
Pathogenesis of Neuropathic Pain
Diagnosis and Treatment /[electronic resource] :edited by Daryl I. Smith, Hai Tran. - 1st ed. 2022. - XVII, 289 p. 42 illus. in color.online resource.
Introduction -- Transitional Neuropathy: Chronification of Acute Pain -- Diabetic Neuropathy -- Complex Regional Pain Syndrome (CRPS) -- Autoimmune Disease-Related Neuropathy -- Methylation Disorder Neuropathy -- Alcohol-Related Neuropathy -- Perfusion-Related Neuropathy -- Uremic Neuropathy -- Chemotherapy Induced Neuropathy -- Antibiotics and Antifungals -- Miscellaneous -- Treatment -- Conclusion.
This comprehensive source on the pathogenic origins of neuropathic pain covers the detailed molecular bases of the currently known neuropathies as classified by their pathogenic origins. Filling a critical need, this book fills the need for a resource on a syndrome that demands improved understanding by clinicians and researchers alike so that treatment options for patients are not categorically limited to a pill or a needle. If the clinician understands the origins of a patients' neuropathic pain, they can work cooperatively toward improving it with tailored therapies that don't create societal diseconomies and that ultimately are effective in helping patients. The book presents in detail the molecular bases of some currently known neuropathies by their pathogenetic origins, allowing clinicians to tailor more specific and more effective treatment regimens for their patients. For basic researchers, this book is a general resource to better direct research on neuropathy-specific molecular mechanisms. The improved understanding of the pathogenesis of neuropathic pain can then be used to develop more specific and more effective manipulations of these pathways. .
ISBN: 9783030914554
Standard No.: 10.1007/978-3-030-91455-4doiSubjects--Topical Terms:
645681
Anesthesiology.
LC Class. No.: RD78.3-87.3
Dewey Class. No.: 617.96
Pathogenesis of Neuropathic Pain = Diagnosis and Treatment /
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Introduction -- Transitional Neuropathy: Chronification of Acute Pain -- Diabetic Neuropathy -- Complex Regional Pain Syndrome (CRPS) -- Autoimmune Disease-Related Neuropathy -- Methylation Disorder Neuropathy -- Alcohol-Related Neuropathy -- Perfusion-Related Neuropathy -- Uremic Neuropathy -- Chemotherapy Induced Neuropathy -- Antibiotics and Antifungals -- Miscellaneous -- Treatment -- Conclusion.
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This comprehensive source on the pathogenic origins of neuropathic pain covers the detailed molecular bases of the currently known neuropathies as classified by their pathogenic origins. Filling a critical need, this book fills the need for a resource on a syndrome that demands improved understanding by clinicians and researchers alike so that treatment options for patients are not categorically limited to a pill or a needle. If the clinician understands the origins of a patients' neuropathic pain, they can work cooperatively toward improving it with tailored therapies that don't create societal diseconomies and that ultimately are effective in helping patients. The book presents in detail the molecular bases of some currently known neuropathies by their pathogenetic origins, allowing clinicians to tailor more specific and more effective treatment regimens for their patients. For basic researchers, this book is a general resource to better direct research on neuropathy-specific molecular mechanisms. The improved understanding of the pathogenesis of neuropathic pain can then be used to develop more specific and more effective manipulations of these pathways. .
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