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Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb.

紀錄類型:	書目-語言資料,手稿 : Monograph/item
正題名/作者:	Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb./
作者:	Foster, Nicholas N.
面頁冊數:	1 online resource (30 pages)
附註:	Source: Masters Abstracts International, Volume: 68-11.
Contained By:	Masters Abstracts International68-11.
標題:	Behavioral psychology. -
電子資源:	click for full text (PQDT)
ISBN:	9781109798111

Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb.
Foster, Nicholas N.

Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb. - 1 online resource (30 pages)

Source: Masters Abstracts International, Volume: 68-11.

Thesis (M.A.)--University of Southern California, 2006.

Includes bibliographical references

Arginine vasopressin (AVP) is a neuropeptide known to influence many rodent social behaviors. Experiment 1 assessed the effects on sexual behavior of AVP (1 ng) infusions into the olfactory bulbs of male rats, finding that AVP significantly inhibited the number of intromissions displayed compared to control animals. Experiment 2 sought to expand upon these findings by comparing groups receiving injections of AVP, a V1b vasopressin receptor antagonist, both AVP and the antagonist (1 ng in all cases), or vehicle. Additionally, immediately after testing, brain tissue samples were collected and were assayed for c-Fos expression in the medial amygdala. AVP alone and paired with the antagonist caused significant reductions in intromission. All drug-treated groups expressed significantly less c-Fos than controls. A model of olfactory processing is proposed whereby the antagonist depresses the function of mitral cells, the primary olfactory bulb efferents, while AVP activates granule cells, which inhibit mitral cells.

Electronic reproduction.
Ann Arbor, Mich. :
ProQuest,
2024

Mode of access: World Wide Web

ISBN: 9781109798111Subjects--Topical Terms:

1179418
Behavioral psychology.
Index Terms--Genre/Form:

554714
Electronic books.

Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb.
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502 $a Thesis (M.A.)--University of Southern California, 2006.
504 $a Includes bibliographical references
520 $a Arginine vasopressin (AVP) is a neuropeptide known to influence many rodent social behaviors. Experiment 1 assessed the effects on sexual behavior of AVP (1 ng) infusions into the olfactory bulbs of male rats, finding that AVP significantly inhibited the number of intromissions displayed compared to control animals. Experiment 2 sought to expand upon these findings by comparing groups receiving injections of AVP, a V1b vasopressin receptor antagonist, both AVP and the antagonist (1 ng in all cases), or vehicle. Additionally, immediately after testing, brain tissue samples were collected and were assayed for c-Fos expression in the medial amygdala. AVP alone and paired with the antagonist caused significant reductions in intromission. All drug-treated groups expressed significantly less c-Fos than controls. A model of olfactory processing is proposed whereby the antagonist depresses the function of mitral cells, the primary olfactory bulb efferents, while AVP activates granule cells, which inhibit mitral cells.
533 $a Electronic reproduction. $b Ann Arbor, Mich. : $c ProQuest, $d 2024
538 $a Mode of access: World Wide Web
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