國立虎尾科技大學 |

Cardiomyocytes – Active Players in Cardiac Disease

Record Type:	Language materials, printed : Monograph/item
Title/Author:	Cardiomyocytes – Active Players in Cardiac Disease/ edited by Klaus-Dieter Schlüter.
other author:	Schlüter, Klaus-Dieter.
Description:	XV, 278 p. 40 illus., 13 illus. in color.online resource. :
Contained By:	Springer Nature eBook
Subject:	Cell biology. -
Online resource:	https://doi.org/10.1007/978-3-319-31251-4
ISBN:	9783319312514

Cardiomyocytes – Active Players in Cardiac Disease
Cardiomyocytes – Active Players in Cardiac Disease[electronic resource] /edited by Klaus-Dieter Schlüter. - 1st ed. 2016. - XV, 278 p. 40 illus., 13 illus. in color.online resource.

Cardiomyocytes: Function and Regeneration -- 1.Ways to study the biology of cardiomyocytes -- 2.Cardiomyocytes: Function and Regeneration -- 3.Excitation-contraction coupling of cardiomyocytes -- 4.Cardiac metabolism and energetic control -- 5.Endogenous mechanisms for regulating myocardial contractility -- Cardiomyocytes in ageing, disease, and protection -- 6.Growth regulation of cardiomyocytes: Control of cell size and its role in cardiac hypertrophy -- 7.Protein Degradation in Cardiomyocytes – Target Proteins and clinical consequences -- 8.Ischemia and Reperfusion -- 9.Mechanisms of cardiac cell death -- 10.Oxidative stress and nitrosative stress.

This book summarizes our current understanding about the biology and patho-biology of cardiomyocytes and depicts common techniques for the study of these cells. The book is divided into two parts; the first part provides insight into role and function of cardiomyocytes under normal conditions and describes embryogenesis and differentiation, in the second part the role of cardiomyocytes in aging and disease is discussed and cellular responses under stress conditions illustrated. Cardiomyocytes represent the main mass of the heart, and cellular malfunction directly modifies heart function leading to subsequent heart failure. As such, cardiomyocytes are causative involved in the main reasons of heart failure, such as post-infarct remodeling, hypertensive heart disease, idiopathic heart failure, and interactions with other co-morbidities such as diabetes. On the other hand, cardiomyocytes are necessarily target of therapy. Therefore, a precise understanding of cardiomyocytes biology is a pre-requisite for proper disease treatment and evidence based medicine. The book is written for cell biologists, pharmacologists and biomedical researchers specialized in cardiac and vascular biology.

ISBN: 9783319312514

Standard No.: 10.1007/978-3-319-31251-4doiSubjects--Topical Terms:

1253486
Cell biology.

LC Class. No.: QH573-671

Dewey Class. No.: 571.6

Cardiomyocytes – Active Players in Cardiac Disease
LDR:03237nam a22003975i 4500 001 978694
003 DE-He213
005 20200706133118.0
007 cr nn 008mamaa
008 201211s2016 gw | s |||| 0|eng d
020 $a 9783319312514 $9 978-3-319-31251-4
024 7 $a 10.1007/978-3-319-31251-4 $2 doi
035 $a 978-3-319-31251-4
050 4 $a QH573-671
072 7 $a PSF $2 bicssc
072 7 $a SCI049000 $2 bisacsh
072 7 $a PSF $2 thema
082 0 4 $a 571.6 $2 23
245 1 0 $a Cardiomyocytes – Active Players in Cardiac Disease $h [electronic resource] / $c edited by Klaus-Dieter Schlüter.
250 $a 1st ed. 2016.
264 1 $a Cham : $b Springer International Publishing : $b Imprint: Springer, $c 2016.
300 $a XV, 278 p. 40 illus., 13 illus. in color. $b online resource.
336 $a text $b txt $2 rdacontent
337 $a computer $b c $2 rdamedia
338 $a online resource $b cr $2 rdacarrier
347 $a text file $b PDF $2 rda
505 0 $a Cardiomyocytes: Function and Regeneration -- 1.Ways to study the biology of cardiomyocytes -- 2.Cardiomyocytes: Function and Regeneration -- 3.Excitation-contraction coupling of cardiomyocytes -- 4.Cardiac metabolism and energetic control -- 5.Endogenous mechanisms for regulating myocardial contractility -- Cardiomyocytes in ageing, disease, and protection -- 6.Growth regulation of cardiomyocytes: Control of cell size and its role in cardiac hypertrophy -- 7.Protein Degradation in Cardiomyocytes – Target Proteins and clinical consequences -- 8.Ischemia and Reperfusion -- 9.Mechanisms of cardiac cell death -- 10.Oxidative stress and nitrosative stress.
520 $a This book summarizes our current understanding about the biology and patho-biology of cardiomyocytes and depicts common techniques for the study of these cells. The book is divided into two parts; the first part provides insight into role and function of cardiomyocytes under normal conditions and describes embryogenesis and differentiation, in the second part the role of cardiomyocytes in aging and disease is discussed and cellular responses under stress conditions illustrated. Cardiomyocytes represent the main mass of the heart, and cellular malfunction directly modifies heart function leading to subsequent heart failure. As such, cardiomyocytes are causative involved in the main reasons of heart failure, such as post-infarct remodeling, hypertensive heart disease, idiopathic heart failure, and interactions with other co-morbidities such as diabetes. On the other hand, cardiomyocytes are necessarily target of therapy. Therefore, a precise understanding of cardiomyocytes biology is a pre-requisite for proper disease treatment and evidence based medicine. The book is written for cell biologists, pharmacologists and biomedical researchers specialized in cardiac and vascular biology.
650 0 $a Cell biology. $3 1253486
650 0 $a Cardiology. $3 593957
650 0 $a Human physiology. $3 636012
650 0 $a Pharmaceutical technology. $3 557391
650 1 4 $a Cell Biology. $3 593889
650 2 4 $a Human Physiology. $3 668349
650 2 4 $a Pharmaceutical Sciences/Technology. $3 768561
700 1 $a Schlüter, Klaus-Dieter. $e editor. $4 edt $4 http://id.loc.gov/vocabulary/relators/edt $3 1272022
710 2 $a SpringerLink (Online service) $3 593884
773 0 $t Springer Nature eBook
776 0 8 $i Printed edition: $z 9783319312491
776 0 8 $i Printed edition: $z 9783319312507
776 0 8 $i Printed edition: $z 9783319809991
856 4 0 $u https://doi.org/10.1007/978-3-319-31251-4
912 $a ZDB-2-SBL
912 $a ZDB-2-SXB
950 $a Biomedical and Life Sciences (SpringerNature-11642)
950 $a Biomedical and Life Sciences (R0) (SpringerNature-43708)