國立虎尾科技大學 |

Social Stress Reactivity : = Links with Early Environmental Instability and Adolescent Anxiety.

紀錄類型:	書目-語言資料,手稿 : Monograph/item
正題名/作者:	Social Stress Reactivity :/
其他題名:	Links with Early Environmental Instability and Adolescent Anxiety.
作者:	Fry, Cassidy.
面頁冊數:	1 online resource (154 pages)
附註:	Source: Dissertations Abstracts International, Volume: 85-11, Section: B.
Contained By:	Dissertations Abstracts International85-11B.
標題:	Child development. -
電子資源:	click for full text (PQDT)
ISBN:	9798382648842

Social Stress Reactivity : = Links with Early Environmental Instability and Adolescent Anxiety.
Fry, Cassidy.

Social Stress Reactivity :Links with Early Environmental Instability and Adolescent Anxiety. - 1 online resource (154 pages)

Source: Dissertations Abstracts International, Volume: 85-11, Section: B.

Thesis (Ph.D.)--The Pennsylvania State University, 2023.

Includes bibliographical references

Although developmental increases in social sensitivity across adolescence are likely adaptive for aiding in the successful navigation of their changing social landscape, some teens might be placed at greater risk for internalizing symptom development (Pfeifer & Allen, 2021). Two studies within the present dissertation seek to better understand for whom, and in what social and developmental contexts, HPA responsivity to social stress might be associated with elevated anxiety in adolescence. Both studies drew from an epidemiological sample of 1292 diverse, lower-income families from rural communities, followed repeatedly from infancy through adolescence.Study 1 focused on developmental unpredictability within the home environment, measuring cumulative household chaos across early life, as well as wave-to-wave fluctuations in caregiver support from infancy through adolescence. Adolescent HPA functioning was additionally examined as, 1) a potential mechanism by which the early environment influenced later anxiety symptoms, and 2) a child-level marker of biological sensitivity that modulated the effects of the environment on anxiety. Results indicated that lability in caregiver support from infancy through adolescence was associated with lesser cortisol reactivity to social stress, though cortisol did not significantly mediate the effects of caregiver support or household chaos on anxiety symptoms. However, for those who displayed greater cortisol reactivity to social stress or lower cortisol at baseline, greater early household disorganization was associated with elevated anxiety. Effects were buffered for those who displayed lesser cortisol reactivity or higher cortisol levels at rest. These results are partly consistent with the biological sensitivity to context framework, highlighting certain patterns of HPA functioning that might increase children's susceptibility to ongoing or future stressors - or that might impact regulatory abilities - within their surrounding environment.Study 2 focused on salient contexts within adolescence, examining the association between concurrent cortisol functioning and anxiety, and the moderating role of peer problems and perceived pubertal stage. Results indicated no direct association between cortisol and anxiety, emphasizing the importance of incorporating developmental and contextual factors when considering the effects of physiological functioning on behavior. Lesser cortisol reactivity was associated with higher trait anxiety for adolescents who reported an earlier perceived pubertal stage. Conversely, greater reactivity was associated with higher risk for a social anxiety diagnosis for those who reported a mid-perceived pubertal stage. Trait anxiety symptoms might be more enduring, such that cortisol hyporeactivity could reflect physiological burnout within the context of developmentally less mature coping skills. However, for those who view themselves as undergoing physical pubertal changes, high physiological sensitivity to social stress might be a more potent risk factor for elevated social anxiety. Overall, these findings point to the complex role of pubertal maturation in anxiety symptom development.

Electronic reproduction.
Ann Arbor, Mich. :
ProQuest,
2024

Mode of access: World Wide Web

ISBN: 9798382648842Subjects--Topical Terms:

554713
Child development.
Index Terms--Genre/Form:

554714
Electronic books.

Social Stress Reactivity : = Links with Early Environmental Instability and Adolescent Anxiety.
LDR:04605ntm a22004217 4500 001 1152164
005 20241122094142.5
006 m o d
007 cr mn ---uuuuu
008 250605s2023 xx obm 000 0 eng d
020 $a 9798382648842
035 $a (MiAaPQ)AAI31085768
035 $a (MiAaPQ)PennState21307cmf338
035 $a AAI31085768
040 $a MiAaPQ $b eng $c MiAaPQ $d NTU
100 1 $a Fry, Cassidy. $3 1479063
245 1 0 $a Social Stress Reactivity : $b Links with Early Environmental Instability and Adolescent Anxiety.
264 0 $c 2023
300 $a 1 online resource (154 pages)
336 $a text $b txt $2 rdacontent
337 $a computer $b c $2 rdamedia
338 $a online resource $b cr $2 rdacarrier
500 $a Source: Dissertations Abstracts International, Volume: 85-11, Section: B.
500 $a Advisor: Kopp, Lisa.
502 $a Thesis (Ph.D.)--The Pennsylvania State University, 2023.
504 $a Includes bibliographical references
520 $a Although developmental increases in social sensitivity across adolescence are likely adaptive for aiding in the successful navigation of their changing social landscape, some teens might be placed at greater risk for internalizing symptom development (Pfeifer & Allen, 2021). Two studies within the present dissertation seek to better understand for whom, and in what social and developmental contexts, HPA responsivity to social stress might be associated with elevated anxiety in adolescence. Both studies drew from an epidemiological sample of 1292 diverse, lower-income families from rural communities, followed repeatedly from infancy through adolescence.Study 1 focused on developmental unpredictability within the home environment, measuring cumulative household chaos across early life, as well as wave-to-wave fluctuations in caregiver support from infancy through adolescence. Adolescent HPA functioning was additionally examined as, 1) a potential mechanism by which the early environment influenced later anxiety symptoms, and 2) a child-level marker of biological sensitivity that modulated the effects of the environment on anxiety. Results indicated that lability in caregiver support from infancy through adolescence was associated with lesser cortisol reactivity to social stress, though cortisol did not significantly mediate the effects of caregiver support or household chaos on anxiety symptoms. However, for those who displayed greater cortisol reactivity to social stress or lower cortisol at baseline, greater early household disorganization was associated with elevated anxiety. Effects were buffered for those who displayed lesser cortisol reactivity or higher cortisol levels at rest. These results are partly consistent with the biological sensitivity to context framework, highlighting certain patterns of HPA functioning that might increase children's susceptibility to ongoing or future stressors - or that might impact regulatory abilities - within their surrounding environment.Study 2 focused on salient contexts within adolescence, examining the association between concurrent cortisol functioning and anxiety, and the moderating role of peer problems and perceived pubertal stage. Results indicated no direct association between cortisol and anxiety, emphasizing the importance of incorporating developmental and contextual factors when considering the effects of physiological functioning on behavior. Lesser cortisol reactivity was associated with higher trait anxiety for adolescents who reported an earlier perceived pubertal stage. Conversely, greater reactivity was associated with higher risk for a social anxiety diagnosis for those who reported a mid-perceived pubertal stage. Trait anxiety symptoms might be more enduring, such that cortisol hyporeactivity could reflect physiological burnout within the context of developmentally less mature coping skills. However, for those who view themselves as undergoing physical pubertal changes, high physiological sensitivity to social stress might be a more potent risk factor for elevated social anxiety. Overall, these findings point to the complex role of pubertal maturation in anxiety symptom development.
533 $a Electronic reproduction. $b Ann Arbor, Mich. : $c ProQuest, $d 2024
538 $a Mode of access: World Wide Web
650 4 $a Child development. $3 554713
650 4 $a Physiology. $3 673386
650 4 $a Behavior. $3 582559
650 4 $a Hormones. $3 676542
650 4 $a Stress response. $3 1476674
650 4 $a Psychopathology. $3 647313
650 4 $a Metabolism. $3 581368
650 4 $a Psychobiology. $3 655331
650 4 $a Emotions. $3 560966
650 4 $a System theory. $3 566168
650 4 $a Homeostasis. $3 1020771
650 4 $a Motivation. $3 655186
650 4 $a Stress. $2 gtt $3 788293
650 4 $a Coping. $3 1469392
650 4 $a Mental health. $3 564038
650 4 $a Caregivers. $3 812777
650 4 $a Self esteem. $3 1437698
650 4 $a Households. $3 892657
650 4 $a Teenagers. $3 558250
650 4 $a Clinical psychology. $3 649607
650 4 $a Developmental psychology. $3 557458
650 4 $a Endocrinology. $3 645497
650 4 $a Neurosciences. $3 593561
650 4 $a Personality psychology. $3 1180475
650 4 $a Systems science. $3 1148479
655 7 $a Electronic books. $2 local $3 554714
690 $a 0349
690 $a 0347
690 $a 0719
690 $a 0622
690 $a 0620
690 $a 0409
690 $a 0317
690 $a 0625
690 $a 0790
710 2 $a ProQuest Information and Learning Co. $3 1178819
710 2 $a The Pennsylvania State University. $3 845556
773 0 $t Dissertations Abstracts International $g 85-11B.
856 4 0 $u http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=31085768 $z click for full text (PQDT)